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Myeloid-Cell Protein Tyrosine Phosphatase-1B Deficiency in Mice Protects Against High-Fat Diet and Lipopolysaccharide-Induced Inflammation, Hyperinsulinemia, and Endotoxemia Through an IL-10 STAT3-Dependent Mechanism

dc.contributor.authorGrant, Louise
dc.contributor.authorShearer, Kirsty D.
dc.contributor.authorCzopek, Alicja
dc.contributor.authorLees, Emma K.
dc.contributor.authorOwen, Carl
dc.contributor.authorAgouni, Abdelali
dc.contributor.authorWorkman, James
dc.contributor.authorMartin-Granados, Cristina
dc.contributor.authorForrester, John V.
dc.contributor.authorWilson, Heather M
dc.contributor.authorMody, Nimesh
dc.contributor.authorDelibegovic, Mirela
dc.date.accessioned2015-05-27T12:32:03Z
dc.date.available2015-05-27T12:32:03Z
dc.date.issued2014-02
dc.identifier27874798
dc.identifiera4cde92c-26c1-4648-b762-c754ceb5bbab
dc.identifier24186864
dc.identifier24186864
dc.identifier84893128687
dc.identifier.citationGrant , L , Shearer , K D , Czopek , A , Lees , E K , Owen , C , Agouni , A , Workman , J , Martin-Granados , C , Forrester , J V , Wilson , H M , Mody , N & Delibegovic , M 2014 , ' Myeloid-Cell Protein Tyrosine Phosphatase-1B Deficiency in Mice Protects Against High-Fat Diet and Lipopolysaccharide-Induced Inflammation, Hyperinsulinemia, and Endotoxemia Through an IL-10 STAT3-Dependent Mechanism ' , Diabetes , vol. 63 , no. 2 , pp. 456-470 . https://doi.org/10.2337/db13-0885en
dc.identifier.issn0012-1797
dc.identifier.otherORCID: /0000-0002-1407-7274/work/83704303
dc.identifier.otherORCID: /0000-0002-2215-7952/work/147568334
dc.identifier.urihttp://hdl.handle.net/2164/4556
dc.format.extent15
dc.format.extent2436020
dc.language.isoeng
dc.relation.ispartofDiabetesen
dc.subjectSDG 3 - Good Health and Well-beingen
dc.subjectR Medicineen
dc.subject.lccRen
dc.titleMyeloid-Cell Protein Tyrosine Phosphatase-1B Deficiency in Mice Protects Against High-Fat Diet and Lipopolysaccharide-Induced Inflammation, Hyperinsulinemia, and Endotoxemia Through an IL-10 STAT3-Dependent Mechanismen
dc.typeJournal articleen
dc.contributor.institutionUniversity of Aberdeen.Medical Sciencesen
dc.contributor.institutionUniversity of Aberdeen.Other Applied Health Sciencesen
dc.contributor.institutionUniversity of Aberdeen.Health Services Research Uniten
dc.contributor.institutionUniversity of Aberdeen.Biological Sciencesen
dc.contributor.institutionUniversity of Aberdeen.Biological Sciencesen
dc.contributor.institutionUniversity of Aberdeen.Applied Medicineen
dc.contributor.institutionUniversity of Aberdeen.Medical Sciences - Cardiovascular Groupen
dc.description.statusPeer revieweden
dc.identifier.doi10.2337/db13-0885


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