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dc.contributor.authorLorenz, Alexander
dc.contributor.authorMehats, Alizée
dc.contributor.authorOsman, Fekret
dc.contributor.authorWhitby, Matthew C
dc.date.accessioned2015-01-06T14:58:01Z
dc.date.available2015-01-06T14:58:01Z
dc.date.issued2014-12-16
dc.identifier46010261
dc.identifierf1b462ef-1389-4fe8-b592-2839c818291d
dc.identifier25414342
dc.identifier84927674849
dc.identifier.citationLorenz , A , Mehats , A , Osman , F & Whitby , M C 2014 , ' Rad51/Dmc1 paralogs and mediators oppose DNA helicases to limit hybrid DNA formation and promote crossovers during meiotic recombination ' , Nucleic Acids Research , vol. 42 , no. 22 , pp. 13723-13735 . https://doi.org/10.1093/nar/gku1219en
dc.identifier.issn0305-1048
dc.identifier.otherORCID: /0000-0003-1925-3713/work/15710171
dc.identifier.urihttp://hdl.handle.net/2164/4142
dc.descriptionThis is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited. ACKNOWLEDGMENTS We are grateful to J ¨urg Kohli, Ramsay J. McFarlane, Paul Russell, Gerald R. Smith, Walter W. Steiner and the National BioResource Project (NBRP) Japan for providing strains and to C. Bryer for technical assistance. FUNDING Wellcome Trust [090767/Z/09/Z to M.C.W.]; College of Life Sciences and Medicine, University of Aberdeen [to A.L., in part]. Funding for open access charge: Wellcome Trusten
dc.format.extent13
dc.format.extent5384261
dc.language.isoeng
dc.relation.ispartofNucleic Acids Researchen
dc.subjectmeiosisen
dc.subjecthomologous recombinationen
dc.subjectSchizosaccharomyces pombeen
dc.subjectRad51en
dc.subjectDmc1en
dc.subjectDNA helicasesen
dc.subjectR Medicineen
dc.subjectWellcome Trusten
dc.subject090767/Z/09/Zen
dc.subjectSupplementary Dataen
dc.subject.lccRen
dc.titleRad51/Dmc1 paralogs and mediators oppose DNA helicases to limit hybrid DNA formation and promote crossovers during meiotic recombinationen
dc.typeJournal articleen
dc.contributor.institutionUniversity of Aberdeen.Medical Sciencesen
dc.contributor.institutionUniversity of Aberdeen.Institute of Medical Sciencesen
dc.description.statusPeer revieweden
dc.identifier.doi10.1093/nar/gku1219


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