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BPA disrupts meiosis I in oogonia by acting on pathways including cell cycle regulation, meiosis initiation and spindle assembly

dc.contributor.authorLoup, Benoit
dc.contributor.authorPoumerol, Elodie
dc.contributor.authorJouneau, Luc
dc.contributor.authorFowler , Paul A
dc.contributor.authorCotinot, Corinne
dc.contributor.authorMandon-Pepin, Beatrice
dc.date.accessioned2022-08-24T08:57:01Z
dc.date.available2022-08-24T08:57:01Z
dc.date.issued2022-08
dc.identifier215775702
dc.identifiera26dc781-52ff-4ea7-a137-e99641fd1884
dc.identifier35667523
dc.identifier85131919172
dc.identifier.citationLoup , B , Poumerol , E , Jouneau , L , Fowler , P A , Cotinot , C & Mandon-Pepin , B 2022 , ' BPA disrupts meiosis I in oogonia by acting on pathways including cell cycle regulation, meiosis initiation and spindle assembly ' , Reproductive Toxicology , vol. 111 , pp. 166-177 . https://doi.org/10.1016/j.reprotox.2022.06.001en
dc.identifier.issn0890-6238
dc.identifier.otherORCID: /0000-0002-4831-9075/work/114164165
dc.identifier.urihttps://hdl.handle.net/2164/19094
dc.descriptionThis work was supported by funding to PAF and CC from the Wellcome Trust (080388) and the European Community's Seventh Framework Programme under grant agreement no. 212885. (http://www.abdn.ac.uk/reef/). The authors would like to thank INRAE, SAAJ, experimental animal facility (Sciences de l'Animal et de l'Aliment de Jouy), especially Jean-Pierre Albert, Didier Mauchand, and Jean-François Alkombre.en
dc.format.extent11
dc.format.extent5129022
dc.language.isoeng
dc.relation.ispartofReproductive Toxicologyen
dc.subjectfetal ovaryen
dc.subjectorgan cultureen
dc.subjectmeiosisen
dc.subjectoogoniaen
dc.subjectBPAen
dc.subjectsheepen
dc.subjectR Medicineen
dc.subjectWellcome Trusten
dc.subject080388en
dc.subjectEuropean Commissionen
dc.subject212885en
dc.subject.lccRen
dc.titleBPA disrupts meiosis I in oogonia by acting on pathways including cell cycle regulation, meiosis initiation and spindle assemblyen
dc.typeJournal articleen
dc.contributor.institutionUniversity of Aberdeen.Molecular and Cellular Functionen
dc.contributor.institutionUniversity of Aberdeen.Applied Medicineen
dc.contributor.institutionUniversity of Aberdeen.Institute of Medical Sciencesen
dc.description.statusPeer revieweden
dc.identifier.doihttps://doi.org/10.1016/j.reprotox.2022.06.001
dc.identifier.vol111en


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